Monoclonal Anti-UGT1A6 Antibody - High-Quality Targeted Antibody

SKU:
Procurenet
Short description
  • Genetically engineered antibody: High-quality monoclonal antibody produced in mice.
  • Specific target: Binds to UGT1A6 protein for precise detection.
  • Effective research tool: Widely used in immunohistochemistry, western blotting, and flow cytometry.
  • Consistent results: Reliable performance for obtaining accurate data.
Quantity :
  • Procurenet Team Tshim Sha Tsui
    Hong Kong Hong Kong 3 years
Description

The Monoclonal Anti-UGT1A6 Antibody is a high-quality, genetically engineered antibody produced in mice. This monoclonal antibody is specifically designed to target and bind to the UGT1A6 protein, a vital component in various biological processes. Clone 4A10, the antibody's unique identity, ensures its effectiveness and specificity in capturing the UGT1A6 protein.

Key Features:

  • Produced in mice through genetic engineering
  • Specifically designed to bind to the UGT1A6 protein
  • Arrives in a ready-to-use format
  • Exceptional effectiveness and reliability

Composition:

  • Monoclonal antibody (Clone 4A10)
  • Purified immunoglobulin
  • Formulated in a buffered aqueous solution for stability

Applications:

  • Immunohistochemistry
  • Western Blotting
  • Flow Cytometry

The Monoclonal Anti-UGT1A6 Antibody, clone 4A10, is widely recognized for its exceptional quality. It offers valuable insights into the UGT1A6 protein's role in various biological phenomena and is an invaluable tool for researchers in various scientific disciplines.

For more information on safety and handling precautions, please refer to the Material Safety Data Sheet (MSDS) provided. Additionally, related peer-reviewed papers and technical documents are available for further exploration of the antibody's applications and potential uses.

Choose the Monoclonal Anti-UGT1A6 Antibody, clone 4A10, for superior results in your research or experimental endeavors. Order now and unlock the potential this powerful antibody holds.

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